The scientists know that we need sleep, but they put ‘t know exactly why, and they ‘ve be rather naive about the way in which the brain decides when to sleep.

Part of the mystery was put to rest. A new study of the mice notes that the cells of the brain invited the fuel of astrocytes to recommend it to sleep by the release of adenosine, chemicals known to have sleep-to induce the effects which can be prevented by cafeine.

The more one person or an animal is waked up long, the more to recommend it to sleep becomes is strong. This is known like pressure of sleep. The former studies indicated adenosine like a release for the pressure of sleep. The chemicals accumulate in the brain during the alarm clock of the hours, helping thereafter to stimulate the single models of the activity of brain which occur during the sleep.

The new found study adenosine of the astrocytes rule clearly the pressure of sleep, member of the team says Michael Halassa of study of Medical school of university of tufts to Boston.

It is the first time that a not-neuronal cell in the brain was shown with the behavior of influence, Halassa said. At the difference of the neurons, the astrocytes do not put fire at the electric transients, and they are often considered as simply cells of support.

This research could carry out to improve of drugs to induce the sleep when it is necessary, and for staving with far the sleep when it is dangerous, said Merrill Mitler of the national institute of the neurological disorders and the race, part of the National Institutes Health funded organization.

Research is today detailed in the journal of neuron .

The scientists reversed a genetic switch in the mice to block the release of adenosine and other chemicals of the astrocytes. The researchers then deprived the mice of the sleep for short periods, and evaluated them with the behavioral tests and by recording the activity of brain.

The mice subjected to the genetic blockade showed less pressure of sleep than mice of order. Following deprivation of sleep, they did not have need for sleep as much compensatory, and during the phases early of the sleep, they had models of compatible activity of brain to low pressure of sleep.

Other studies of the mechanism recently discovered of sleep in the mice could help to indicate why people need sleep of the whole, the scientists said.

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